Abstract Objective To identify structural and neurochemical properties that underlie functional connectivity impairments of the primary motor cortex (PMC) and how these relate to clinical findings
Abstract Amyotrophic lateral sclerosis (ALS) is a fatal disease characterized by aberrant alternative splicing (AS). Nuclear loss and cytoplasmic accumulation of the splicing factor TDP-43
An endogenous human retrovirus called HERV-K is expressed at higher levels in the brains of amyotrophic lateral sclerosis (ALS) patients than in those of controls,
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