Abstract Although circumstantial evidence supports enhanced Toll-like receptor 7 (TLR7) signalling as a mechanism of human systemic autoimmune disease1,2,3,4,5,6,7, evidence of lupus-causing TLR7 gene variants is lacking. Here
Cigarette smoke contributes to epigenetic and genetic changes in lung cancer. But whether and how these alterations interact in lung cancer initiation has remained unclear.
Passenger mutations unwanted alterations that accompany intentional genetic modifications can confound experiments using certain types of transgenic mice. According to an analysis published today (July
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