Abstract The transferability and clinical value of genetic risk scores (GRSs) across populations remain limited due to an imbalance in genetic studies across ancestrally diverse
Abstract Embryonic development is dictated by tight regulation of DNA replication, cell division and differentiation. Mutations in DNA repair and replication genes disrupt this equilibrium,
Highlights •CaMKIIα phosphorylates GluN2A at Ser-1459 in response to glycine stimulation •CaMKIIα and SNX27 are required for the glycine-induced increase in surface GluN2A •GluN2A Ser-1459
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